From:  Targeted therapeutic management of diabetes using phytoconstituents: molecular mechanisms, evidence map (2015–2025), and translational outlook

 We extracted biomarker data associated with each pathway to contextualize mechanistic evidence (e.g., IRS-1, GLUT4 for PI3K/AKT; ACC phosphorylation for AMPK).

VariableDescriptionSource example (Entry #)
Plant/PhytoconstituentFicus deltoidea, Curcumin#1, #101
Study typeIn vitro, in vivo, in silico, or combined#6 (in vitro), #54 (in vivo)
Diabetes modelSTZ rats, HFD mice, computational targets#1 (STZ-NA rats), #2 (HFD)
Molecular targetPI3K/AKT, PTP1B, PPARγ, α-glucosidase#12 (IRS-1/AKT), #102 (α-amylase)
Key outcomes↓ Glucose, ↑ insulin sensitivity, and ↓ inflammation#3 (↓ glucose), #46 (↑ insulin sensitivity)

ACC: acetyl-CoA carboxylase; AMPK: AMP-activated protein kinase; GLUT4: glucose transporter 4; HFD: high-fat diet; IRS-1: insulin receptor substrate 1; PI3K/AKT: phosphoinositide 3-kinase/protein kinase B; PPARγ: peroxisome proliferator-activated receptor gamma; PTP1B: protein tyrosine phosphatase 1B; STZ: streptozotocin.