From:  Glucagon-like peptide 1 receptor agonists in cardiovascular-kidney-metabolic syndrome: a review

 Clinical Trials of glucagon-like peptide-1 receptor agonists (GLP-1Ras) in cardiometabolic diseases.

DrugsName of the studyType of the studyResearch areaParticipantsKey findingsConclusionReference
SemaglutideSTEP 1 clinical trialsRandomized, double-blind, placebo-controlled, multicenter, phase 3 trialOverweight and obesity1,961 patients with BMI ≥ 30 (or ≥ 27 with ≥ 1 weight-related comorbidity) and without diabetesSemaglutide 2.4 mg once weekly led to a mean weight loss of 14.9% (vs 2.4% with placebo) and a mean weight reduction of 15.3 kg (vs 2.6 kg with placebo) over 68 weeks.Semaglutide 2.4 mg once weekly achieved significant weight loss in participants with overweight or obesity[14]
SemaglutideSTEP 2 clinical trialsRandomized, double-blind, double-dummy, placebo-controlled, multicenter, superiority, phase 3 trialOverweight and obesity
T2DM
1,210 patients with BMI ≥ 27 and T2DMSemaglutide 2.4 mg once weekly resulted in a mean weight loss of 9.6% (vs 3.4% with placebo)Semaglutide 2.4 mg once weekly achieved significant weight loss in adults with overweight or obesity and T2DM[44]
SemaglutideSTEP 4 clinical trialsRandomized, double-blind, placebo-controlled withdrawal study, phase 3 trialOverweight and obesity803 patients with BMI ≥ 30 (or ≥ 27 with ≥ 1 weight-related comorbidity) and without diabetesContinued semaglutide treatment resulted in sustained weight loss, with a mean weight change of –7.9% from week 20 to week 68, compared to +6.9% with placeboContinued semaglutide treatment sustained weight loss over 48 weeks[15]
Semaglutide
liraglutide
STEP 8 clinical trialsRandomized, double-blind, placebo-controlled open-label, phase 3b trialOverweight and obesity338 patients with BMI ≥ 30 (or ≥ 27 with ≥ 1 weight-related comorbidity) and without diabetesSemaglutide (2.4 mg) led to a mean weight loss of 15.8%, compared with 6.4% for liraglutide (3.0 mg) at 68 weeksSemaglutide resulted in significantly greater weight loss than liraglutide[45]
TirzepatideSURMOUNT-1 clinical trialsRandomized, double-blind, placebo-controlled, phase 3 trialOverweight and obesity2,539 patients with BMI ≥ 30 (or ≥ 27 with ≥ 1 weight-related comorbidity) and without diabetesAt week 72, tirzepatide (5 mg, 10 mg, 15 mg) led to mean weight loss of 15.0%, 19.5%, and 20.9%, respectively, compared to 3.1% with placebo.Tirzepatide achieved significant and sustained weight loss in participants with obesity[46]
TirzepatideSURMOUNT-2 clinical trialsRandomized, double-blind, multicentre, placebo-controlled, phase 3 trialOverweight and obesity
T2DM
1,514 adults with BMI ≥ 27 and glycated hemoglobin (HbA1c) of 7–10% (53–86 mmol/mol)At week 72, tirzepatide (10 mg, 15 mg) achieved mean weight loss of 12.8%, 14.7% vs 3.2% with placebo.Tirzepatide resulted in significant weight loss in adults with obesity and T2DM[47]
Semaglutide Cagrilintide (Dual Amylin–Calcitonin Receptor Agonist)REDEFINE 1 clinical trialsRandomized, multicenter, double-blind, placebo-controlled, active-controlled, phase 3a trialOverweight and obesity3,417 patients with BMI ≥ 30 (or ≥ 27 with ≥ 1 weight-related comorbidity) and without diabetesAt week 68, the combination of semaglutide 2.4 mg and cagrilintide 2.4 mg (CagriSema) led to a mean weight loss of –20.4% vs –3.0% with placebo; 91.9% of participants receiving CagriSema achieved at least a 5% weight reductionCagriSema significantly reduced body weight in adults with overweight or obesity[49]
Semaglutide Cagrilintide (Dual Amylin–Calcitonin Receptor Agonist)REDEFINE 2 clinical trialsRandomized, international, double-blind, placebo-controlled, phase 3 trialOverweight and obesity
T2DM
1,206 patients with BMI ≥ 27, a HbA1c level of 7 to 10%, and T2DMAt week 68, cagrilintide-semaglutide (2.4 mg each) led to a mean weight loss of –13.7% vs –3.4% with placebo; 86.5% of participants in the CagriSema group achieved at least a 5% weight reductionCagriSema significantly reduced body weight in adults with obesity and T2DM[50]
MazdutideGLORY-1 clinical trialsRandomized, double-blind, parallel-group, phase 3 trialOverweight and obesity610 patients with BMI ≥ 28 (or ≥ 24 with ≥ 1 weight-related comorbidity)At week 48, Mazdutide (4mg, 6mg) achieved mean weight loss of 11.00% and 14.01% vs 0.30% with placeboMazdutide significantly reduced body weight in adults with overweight or obesity and was well tolerated[57]
Maridebart CafraglutideMARITIME-1
clinical trials
Randomized, double-blind, parallel-group, phase 2 trialOverweight and obesity
T2DM
465 patients with obesity and 127 individuals with obesity and diabetesAt week 52, Maridebart-cafraglutide achieved mean weight loss of –12.3% to –16.2% in the obesity cohort and –8.4% to –12.3% in the obesity-diabetes cohort, compared to –2.5% and –1.7% with placeboMaridebart cafraglutide significantly reduced body weight in participants with obesity, with or without T2DM[58]
EcnoglutideSLIMMER clinical trialsRandomized, double-blind, multicenter, placebo-controlled, phase 3 trialOverweight or obesity882 patients with BMI ≥ 28 (or ≥ 24 with ≥ 1 weight-related comorbidity) without diabetesAt week 40, mean weight loss was –9.1% (1.2 mg), –10.9% (1.8 mg), and –13.2% (2.4 mg) vs 0.1% (placebo). At least 5% weight reduction was achieved by 77% (1.2 mg), 84% (1.8 mg), and 87% (2.4 mg) vs 16% (placebo).Ecnoglutide significantly reduced weight with a favorable safety profile[59]
Semaglutide VS DulaglutideSUSTAIN 7 clinical trialsRandomized, open-label, parallel-group, phase 3b trialT2DM1,201 patients aged 18 years or older with T2DM with HbA1c 7.0–10.5% (53.0–91.0 mmol/mol) on metformin monotherapyAt week 40, semaglutide 0.5 mg reduced HbA1c by 1.5% vs 1.1% with dulaglutide 0.75 mg, and semaglutide 1.0 mg reduced HbA1c by 1.8% vs 1.4% with dulaglutide 1.5 mg. Semaglutide led to weight loss of –4.6 kg with 0.5 mg vs –2.3 kg with 0.75 mg dulaglutide and –6.5 kg with 1.0 mg vs –3.0 kg with 1.5 mg dulaglutide.Semaglutide was superior to dulaglutide in improving glycemic control and reducing body weight, with a similar safety profile[22]
SemaglutideSTEP-HFpEF DM clinical trialsRandomized, international, double-blind, placebo-controlled, phase 3 trialCVD
Obesity
T2DM
616 patients who had HFpEF, BMI ≥ 30 kg/m2 and T2DMSemaglutide led to a mean KCCQ-CSS improvement of 13.7 points and a mean weight loss of 9.8%, compared to 6.4 points and 3.4% with placeboSemaglutide resulted in greater reductions in symptoms and weight loss than placebo in patients with HFpEF, obesity, and T2DM[60]
SemaglutideSTEP-HFpEF clinical trialsRandomized, international, double-blind, placebo-controlled, phase 3 trialObesity
CVD
529 patients who had HFpEF and BMI ≥ 30 kg/m2Semaglutide (2.4 mg) significantly improved symptoms and physical limitations, with a mean KCCQ-CSS increase of 16.6 points and a mean weight loss of 13.3%, compared to 8.7 points and 2.6% with placeboSemaglutide (2.4 mg) led to greater improvements in symptoms, physical limitations, and weight loss than placebo in patients with HFpEF and obesity[61]
SemaglutideSELECT clinical trialsRandomized, double-blind, multicenter, placebo-controlled, event-driven superiority trialCVD
Overweight or obesity
17,604 patients aged 45 or older with preexisting CVD and BMI ≥ 27 kg/m2 without diabetesPrimary cardiovascular events occurred in 6.5% of the semaglutide group vs 8.0% of the placebo group (HR 0.80; 95% CI 0.72–0.90; P < 0.001)Semaglutide reduced cardiovascular events in patients with CVD and overweight/obesity without diabetes[9]
DulaglutideREWIND clinical trialsRandomized, double-blind, multicenter, placebo-controlled trialCVD
T2DM
9,901 patients aged 45 or older with T2DM who had either a previous cardiovascular event or cardiovascular risk factorsDulaglutide reduced major adverse cardiovascular events (MACE) by 12% in patients with T2DM and cardiovascular risk factors. The primary composite outcome occurred in 12.0% of participants in the dulaglutide group vs 13.4% in the placebo group during a median follow-up of 5.4 yearsDulaglutide demonstrated significant cardiovascular protection in patients with T2DM and cardiovascular risk factors[26]
SemaglutideSOUL clinical trialsRandomized, double-blind, placebo-controlled, event-driven, superiority trialCVD
T2DM
CKD
9,650 patients aged 50 years or older with T2DM (HbA1c 6.5%–10.0%) and atherosclerotic CVD, CKD, or bothAfter a mean follow-up of 47.5 months, the incidence of major adverse cardiovascular events (MACE) was 12.0% in the semaglutide group and 13.8% in the placebo groupSemaglutide significantly reduced the risk of major adverse cardiovascular events without increasing the incidence of serious adverse events[62]
SemaglutideFLOW clinical trialsRandomized, double-blind, International, parallel-group, phase 3 trialCKD
T2DM
3,533 patients with T2DM and CKD (defined by an estimated glomerular filtration rate [eGFR] of 50 to 75 mL per minute per 1.73 m2 of body-surface area and a urinary albumin-to-creatinine ratio [with albumin measured in milligrams and creatinine measured in grams] of > 300 and < 5,000 or an eGFR of 25 to < 50 mL per minute per 1.73 m2 and a urinary albumin-to-creatinine ratio of > 100 and < 5,000)Semaglutide reduced the risk of major kidney disease events by 24% and major cardiovascular events by 18%Semaglutide significantly lowered the risk of kidney failure and cardiovascular death in patients with T2DM and CKD[8]
SemaglutideTirzepatide in MASHRandomized, double-blind, multicenter, placebo-controlled, ongoing phase 3 trialMetabolic dysfunction-associated steatohepatitis (MASH)1,197 patients with biopsy-defined MASH and fibrosis stage 2 or 3At week 72, semaglutide resolved steatohepatitis in 62.9% of patients vs 34.3% with placebo and reduced liver fibrosis in 36.8% vs 22.4% with placeboSemaglutide significantly improved liver histology in patients with MASH and moderate to advanced fibrosis[63]

BMI: body mass index; CKD: chronic kidney disease; CVD: cardiovascular disease; HFpEF: heart failure with preserved ejection fraction; T2DM: type 2 diabetes mellitus.