From:  Cellular state transitions in neuroimmune disorders

 Peripheral immune cells infiltrating the central nervous system during neuroinflammation.

Immune cell typeOriginEntry mechanismKey functions of CNSAssociated diseases
Monocyte-derived macrophagesCirculating monocytes originating from bone marrow hematopoiesisMigration across blood-brain barrier through adhesion molecules (ICAM-1, VCAM-1) and chemokine signalingPhagocytosis of cellular debris, production of inflammatory mediators, contribution to tissue remodeling and repairMultiple sclerosis, stroke, traumatic brain injury [183, 184]
CD4+ T cellsPeripheral lymphoid organsAntigen-dependent migration across inflamed vascular endothelium via chemokine-directed traffickingRegulation of immune responses through cytokine production, coordination of inflammatory signalingMultiple sclerosis, viral CNS infections [187]
CD8+ T cellsPeripheral lymphoid organsRecruitment through chemokine gradients and adhesion molecule interactions at the blood-brain barrierCytotoxic responses against infected or damaged cells, antigen-specific immune defenseViral encephalitis, autoimmune neuroinflammation [190, 191]
Regulatory T cells (Tregs)Peripheral immune system, thymus-derived T-cell lineageMigration to inflamed CNS tissue through chemokine-mediated traffickingSuppression of excessive inflammation, modulation of immune responses, support of tissue protection and repairMultiple sclerosis, neurodegenerative disorders [186]
NeutrophilsBone marrow-derived granulocytesRapid recruitment through endothelial adhesion molecules and chemokine signaling during acute inflammationRelease of proteases, reactive oxygen species, and inflammatory mediators; contribution to early inflammatory responsesStroke, traumatic brain injury, infection [188, 192]