The data include the principal molecular mechanisms and functional outcomes. Aβ: amyloid-beta; AD: Alzheimer’s disease; BDNF: brain-derived neurotrophic factor; CAT: catalase; CPC: C-Phycocyanin; CYBB: cytochrome b-245 beta chain; EAE: experimental autoimmune encephalomyelitis; HO-1: heme oxygenase-1; IFN-γ: interferon-γ; IL-6: interleukin-6; MS: multiple sclerosis; NOX2: nicotinamide adenine dinucleotide phosphate oxidase 2; PCB: phycocyanobilin; SOD2: superoxide dismutase 2; TNF-α: tumor necrosis factor-alpha. The arrow pointing upwards (↑) represents an increase, and the arrow pointing downwards (↓) represents a decrease.
Declarations
Acknowledgments
We thank Dr. Ania Cabrales-Rico and Osmany Guirola-Cruz for their technical assistance with the images, which was essential for the successful completion of this manuscript. The authors acknowledge the use of ChatGPT (OpenAI, San Francisco, CA, USA) to assist in improving the readability of the manuscript. All content was critically reviewed and approved by the authors, who take full responsibility for the publication.
Author contributions
SDL and YRÁ: Methodology, Writing—original draft, Formal analysis. GPR: Supervision, Methodology, Writing—review & editing. All authors read and approved the final version of the manuscript and agree to be accountable for all aspects of the work.
Conflicts of interest
The authors declare that they have no conflicts of interest.
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