From:  Hyperuricemia-gout, psoriatic disease, and what to expect from advanced anti-obesity therapies

 Integrated approach to hyperuricemia, gout, and cardiometabolic risk in psoriatic disease.

StepFocusKey actions
1. Risk identification and baseline evaluationPsA patients with ↑ BMI, metabolic comorbidities, or active diseaseAssess SUA, lipids, BP, HbA1c, inflammatory markers, PASI/DAPSA, eGFR, and waist circumference
2. Management of goutIf gout presentStart ULT (allopurinol/febuxostat); SUA target < 6 mg/dL; flare prophylaxis (colchicine/NSAIDs); note: no CV prevention with ULT
3. Weight and metabolic healthObesity and systemic inflammationGLP-1 RAs, tirzepatide + lifestyle; benefits: weight ↓, inflammation ↓, modest SUA ↓
4. Vascular risk managementCV preventionTreat LDL, BP, smoking; statins if high risk (LDL < 70 mg/dL); SGLT2i for DM: SUA ↓, renal and vascular benefit
5. Integrated care approachMultidisciplinaryCoordinate with cardiology/PCP; monitor SUA, weight, inflammation, lipids; combine GLP-1/GIP + traditional medicines
6. Monitoring and follow-upLong-termTrack SUA, weight, PASI/DAPSA; maintain ULT if active gout persists despite weight loss

PsA: psoriatic arthritis; BMI: body mass index; SUA: serum uric acid; BP: blood pressure; HbA1c: glycated hemoglobin; PASI: Psoriasis Area and Severity Index; DAPSA: Disease Activity in Psoriatic Arthritis; eGFR: estimated glomerular filtration rate; ULT: urate-lowering therapy; CV: cardiovascular; GLP-1 RAs: glucagon-like peptide-1 receptor agonists; GIP: glucose-dependent insulinotropic polypeptide; SGLT2i: sodium-glucose cotransporter-2 inhibitors; PCP: primary care physician; ↑: increased; ↓: reduction/diminishing.